The NeuroICU Book (Neurology)

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What is the purpose of invasive neuromonitoring in comatose patients?

Davis PT Collection. Murtagh Collection. About Search. Enable Autosuggest. Previous Chapter. Next Chapter. Multimodality Neuromonitoring. In: Lee K. Lee K Ed. Kiwon Lee. Accessed October 06, Download citation file: RIS Zotero. Reference Manager. Autosuggest Results. Expand All Sections. Jump to a Section Case What is the purpose of invasive neuromonitoring in comatose patients? Which monitoring devices should be placed in this patient? Where should monitoring devices be placed?

The NeuroICU Book by Kiwon Lee (2012, Paperback)

What are the complications of invasive neuromonitoring? When should Pbt o 2 be monitored? What is the meaning of the decrease in Pbt o 2 , and how should the patient be treated? What is the relationship between CO 2 and Pbt o 2? How do you interpret this figure, and what are treatment options? Is an increase in FI o 2 an efficient treatment strategy in patients with low Pbt o 2? Should this patient be transfused?

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Are there other management strategies to increase Pbt o 2? Does optimization of cardiac performance in patients with normal ICP improve oxygenation status? Can a correlational analysis of multimodality parameters be used to assess autoregulatory status?

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Do any of the multimodality parameters allow further insight into the autoregulatory status? Which parameters are routinely measured with MD, and what are the normal values of brain biochemistry?

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What is the relationship between Pbt o 2 and brain metabolism, and what is the most likely cause for the increase in LPR and decreasing levels of brain glucose in this patient? What is a typical multimodality pattern seen during vasospasm? What is the definition of metabolic distress-metabolic crisis, and how does it affect outcome? Our patient was treated with 20 mg IA verapamil. What are the expected hemodynamic changes, and how may IA Ca-channel blockers affect brain homeostasis?

The Neuro ICU Book

What is the relationship between serum glucose and brain glucose, and how should this patient be managed? Are there other factors beside serum glucose influencing brain tissue glucose levels? How should variations in lactate and pyruvate concentrations be interpreted when LPR is not affected? What information could be added by monitoring regional cerebral blood flow? What is the effect of fever on multimodality parameters? Can multimodality monitoring help in the interpretation of questionable EEG findings?

How do you interpret the EEG findings in this woman reference axes indicate mm tracing height and a 1-second time interval. This item may be a floor model or store return that has been used.

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The Neuro ICU Book

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go to link Add to cart. Be the first to write a review About this product. About this product Product Information The first practical, protocol-based guide to the emerging field of neurocritical care The goal of neurological critical care is to rapidly deliver acute medical therapies and appropriate interventions through effective monitoring of both neurological and end organ function.

Combining the latest clinical perspectives in critical care medicine, neurology, and neurosurgery, this comprehensive, evidence-based text standardizes neurocritical care and takes you through the rationale for those standards. Filled with detailed case studies and featuring a question-and-answer format, the book not only builds competency in recognizing acute changes in neurological function, but also addresses all organ insufficiencies and failures, reflecting the real-life challenges in the modern neuro-ICU.

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